Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. On the flip side, our capability to rescue SERT perform in KI midbrain synaptoneurosomes because of the inhibition of FAK signifies elevated FAK signaling downstream from the Pro32Pro33 mutant, as confirmed by enhanced pFAK localization in https://carsonp753ufq5.csublogs.com/profile